The concurrency of edges, quantified by the number of edges that share a common node at a given time point, may be an important determinant of epidemic processes in temporal networks. We propose theoretically tractable Markovian temporal network models in which each edge flips between the active and inactive states in continuous time. The different models have different amounts of concurrency while we can tune the models to share the same statistics of edge activation and deactivation (and hence the fraction of time for which each edge is active) and the structure of the aggregate (i.e., static) network. We analytically calculate the amount of concurrency of edges sharing a node for each model. We then numerically study effects of concurrency on epidemic spreading in the stochastic susceptible-infectious-susceptible and susceptible-infectious-recovered dynamics on the proposed temporal network models. We find that the concurrency enhances epidemic spreading near the epidemic threshold while this effect is small in many cases. Furthermore, when the infection rate is substantially larger than the epidemic threshold, the concurrency suppresses epidemic spreading in a majority of cases. In sum, our numerical simulations suggest that the impact of concurrency on enhancing epidemic spreading within our model is consistently present near the epidemic threshold but modest. The proposed temporal network models are expected to be useful for investigating effects of concurrency on various collective dynamics on networks including both infectious and other dynamics.
Social contact networks underlying epidemic processes in humans and animals are highly dynamic. The spreading of infections on such temporal networks can differ dramatically from spreading on static networks. We theoretically investigate the effects of concurrency, the number of neighbors that a node has at a given time point, on the epidemic threshold in the stochastic susceptible-infected-susceptible dynamics on temporal network models. We show that network dynamics can suppress epidemics (i.e., yield a higher epidemic threshold) when the nodes' concurrency is low, but can also enhance epide…
In this paper, we study the control of the traffic-driven epidemic spreading by immunization strategy. We consider the random, degree-based and betweeness-based immunization strategies, respectively. It is found that the betweeness-based immunization strategy can most effectively prevent the outbreak of traffic-driven epidemic. Besides, we find that the critical number of immune nodes above which epidemic dies out is increased with the enhancement of the spreading rate and the packet-generation rate.
Models of epidemic spreading on complex networks have attracted great attention among researchers in physics, mathematics, and epidemiology due to their success in predicting and controlling scenarios of epidemic spreading in real-world scenarios. To understand the interplay between epidemic spreading and the topology of a contact network, several outstanding theoretical approaches have been developed. An accurate theoretical approach describing the spreading dynamics must take both the network topology and dynamical correlations into consideration at the expense of increasing the complexity o…
The disease spreading on complex networks is studied in SIR model. Simulations on empirical complex networks reveal two specific regimes of disease spreading: local containment and epidemic outbreak. The variables measuring the extent of disease spreading are in general characterized by a bimodal probability distribution. Phase diagrams of disease spreading for empirical complex networks are introduced. A theoretical model of disease spreading on m-ary tree is investigated both analytically and in simulations. It is shown that the model reproduces qualitative features of phase diagrams of dise…
We study the traffic-driven epidemic spreading on scale-free networks with tunable degree distribution. The heterogeneity of networks is controlled by the exponent $γ$ of power-law degree distribution. It is found that the epidemic threshold is minimized at about $γ=2.2$. Moreover, we find that nodes with larger algorithmic betweenness are more likely to be infected. We expect our work to provide new insights into the effect of network structures on traffic-driven epidemic spreading.
The design of routing strategies for traffic-driven epidemic spreading has received increasing attention in recent years. In this paper, we propose an adaptive routing strategy that incorporates topological distance with local epidemic information through a tunable parameter $h$. In the case where the traffic is free of congestion, there exists an optimal value of routing parameter $h$, leading to the maximal epidemic threshold. This means that epidemic spreading can be more effectively controlled by adaptive routing, compared to that of the static shortest path routing scheme. Besides, we fin…
We introduce non-Markovian SIR epidemic spreading model inspired by the characteristics of the COVID-19, by considering discrete- and continuous-time versions. The incubation period, delayed infectiousness and the distribution of the recovery period are modeled with general functions. By taking corresponding choice of these functions, it is shown that the model reduces to the classical Markovian case. The epidemic threshold is analytically determined for arbitrary functions of infectivity and recovery and verified numerically. The relevance of the model is shown by modeling the first wave of t…
We consider a standard \textit{susceptible-infected-susceptible} (SIS) model to study behaviors of steady states of epidemic spreading in small-world networks. Using analytical methods and large scale simulations, we recover the usual epidemic behavior with a critical threshold $λ_{c}$ below which infectious diseases die out. Whereas for the spreading rate $λ$ far above $λ_{c}$, it was found that the density of infected individuals $ρ$ as a function of $λ$ has the property $ρ\approx F(K)(\ln λ- \ln λ_{c})$.
Bipartite networks are composed of two types of nodes and there are no links between nodes of the same type. Thus the study of epidemic spread and control on such networks is relevant to sexually transmitted diseases (STDs). When entire populations of two types cannot be immunized and the effect of immunization is not perfect, we have to consider the targeted immunization with immunization rates. We derive the epidemic thresholds of SIR and SIS models with immunization and illustrate the results with STDs on heterosexual contact networks.
We present a modified \emph{susceptible-infected-susceptible} (SIS) model on complex networks, small-world and scale-free, to study epidemic spreading with the effect of time delay which is introduced to the infected phase. Considering the topology of the network, both uniform and degree-dependent delays are studied during the contagion process. We find that the existence of time delay will enhance both outbreaks and prevalence of infectious diseases in the network.
We study geographical effects on the spread of diseases in lattice-embedded scale-free networks. The geographical structure is represented by the connecting probability of two nodes that is related to the Euclidean distance between them in the lattice. By studying the standard Susceptible-Infected model, we found that the geographical structure has great influences on the temporal behavior of epidemic outbreaks and the propagation in the underlying network: the more geographically constrained the network is, the more smoothly the epidemic spreads.
We investigate the effects of modular and temporal connectivity patterns on epidemic spreading. To this end, we introduce and analytically characterise a model of time-varying networks with tunable modularity. Within this framework, we study the epidemic size of Susceptible-Infected-Recovered, SIR, models and the epidemic threshold of Susceptible-Infected-Susceptible, SIS, models. Interestingly, we find that while the presence of tightly connected clusters inhibit SIR processes, it speeds up SIS diseases. In this case, we observe that heterogeneous temporal connectivity patterns and modular st…
Network properties govern the rate and extent of various spreading processes, from simple contagions to complex cascades. Recently, the analysis of spreading processes has been extended from static networks to temporal networks, where nodes and links appear and disappear. We focus on the effects of "accessibility", whether there is a temporally consistent path from one node to another, and "reachability", the density of the corresponding "accessibility graph" representation of the temporal network. The level of reachability thus inherently limits the possible extent of any spreading process on…
Recent studies on network geometry, a way of describing network structures as geometrical objects, are revolutionizing our way to understand dynamical processes on networked systems. Here, we cope with the problem of epidemic spreading, using the Susceptible-Infected-Susceptible (SIS) model, in simplicial complexes. In particular, we analyze the dynamics of SIS in complex networks characterized by pairwise interactions (links), and three-body interactions (filled triangles, also known as 2-simplices). This higher-order description of the epidemic spreading is analytically formulated using a mi…
The spreading of an infectious disease can trigger human behavior responses to the disease, which in turn plays a crucial role on the spreading of epidemic. In this study, to illustrate the impacts of the human behavioral responses, a new class of individuals, $S^F$, is introduced to the classical susceptible-infected-recovered ($SIR$) model. In the model, $S^F$ state represents that susceptible individuals who take self-initiate protective measures to lower the probability of being infected, and a susceptible individual may go to $S^F$ state with a response rate when contacting an infectious …
Many real networks are embedded in a metric space: the interactions among individuals depend on their spatial distances and usually take place among their nearest neighbors. In this paper, we introduce a modified susceptible-infected-susceptible (SIS) model to study geographical effects on the spread of diseases by assuming that the probability of a healthy individual infected by an infectious one is inversely proportional to the Euclidean distance between them. It is found that geography plays a more important role than hubs in disease spreading: the more geographically constrained the networ…
The study of epidemic spreading on populations of networked individuals has seen recently a great deal of significant progresses. A common point of all past studies is, however, that there is only one peak of infected density in each single epidemic spreading episode. At variance, real data from different cities over the world suggest that, besides a major single peak trait of infected density, a finite probability exists for a pattern made of two (or multiple) peaks. We show that such a latter feature is fully distinctive of a multilayered network of interactions, and reveal that actually a t…
We present a contact-based model to study the spreading of epidemics by means of extending the dynamic message passing approach to temporal networks. The shift in perspective from node- to edge-centric quantities enables accurate modelling of Markovian susceptible-infected-recovered outbreaks on time-varying trees, i.e., temporal networks with a loop-free underlying topology. On arbitrary graphs, the proposed contact-based model incorporates potential structural and temporal heterogeneities of the underlying contact network and improves analytic estimations with respect to the individual-based…
Network properties govern the rate and extent of spreading processes on networks, from simple contagions to complex cascades. Recent advances have extended the study of spreading processes from static networks to temporal networks, where nodes and links appear and disappear. We review previous studies on the effects of temporal connectivity for understanding the spreading rate and outbreak size of model infection processes. We focus on the effects of "accessibility", whether there is a temporally consistent path from one node to another, and "reachability", the density of the corresponding "ac…
Changs in individual behavior often entangles with the dynamic interaction of individuals, which complicates the epidemic process and brings great challenges for the understanding and control of the epidemic. In this work, we consider three kinds of typical behavioral changes in epidemic process that is, self-quarantine of infected individuals, self-protection of susceptible individuals, and social distancing between them. We connect the behavioral changes with individual's social attributes by the activity-driven network with attractiveness. A mean-field theory is established to derive an ana…
can evolve simultaneously. For the information-driven adaptive process, susceptible (infected) individuals who have abilities to recognize the disease would break the links of their infected (susceptible) neighbors to prevent the epidemic from further spreading. Simulation results and numerical analyses based on the pairwise approach indicate that the information-driven adaptive process can not only slow down the speed of epidemic spreading, but can also diminish the epidemic prevalence at the final state significantly. In addition, the disease spreading and information diffusion pattern on th…
Despite extensive work on the interplay between traffic dynamics and epidemic spreading, the control of epidemic spreading by routing strategies has not received adequate attention. In this paper, we study the impact of efficient routing protocol on epidemic spreading. In the case of infinite node-delivery capacity, where the traffic is free of congestion, we find that that there exists optimal values of routing parameter, leading to the maximal epidemic threshold. This means that epidemic spreading can be effectively controlled by fine tuning the routing scheme. Moreover, we find that an incr…
Epidemic spread on networks is one of the most studied dynamics in network science and has important implications in real epidemic scenarios. Nonetheless, the dynamics of real epidemics and how it is affected by the underline structure of the infection channels are still not fully understood. Here we apply the SIR model and study analytically and numerically the epidemic spread on a recently developed spatial modular model imitating the structure of cities in a country. The model assumes that inside a city the infection channels connect many different locations, while the infection channels be…
In this paper, we study into the impact of the preference of an individual for public transport on the spread of infectious disease, through a quantity known as the public mobility. Our theoretical and numerical results based on a constructed model reveal that if the average public mobility of the agents is fixed, an increase in the diversity of the agents' public mobility reduces the epidemic threshold, beyond which an enhancement in the rate of infection is observed. Our findings provide an approach to improve the resistance of a society against infectious disease, while preserving the utili…
We investigate a fermionic susceptible-infected-susceptible model with mobility of infected individuals on uncorrelated scale-free networks with power-law degree distributions $P (k) \sim k^{-γ}$ of exponents $2<γ<3$. Two diffusive processes with diffusion rate $D$ of an infected vertex are considered. In the \textit{standard diffusion}, one of the nearest-neighbors is chosen with equal chance while in the \textit{biased diffusion} this choice happens with probability proportional to the neighbor's degree. A non-monotonic dependence of the epidemic threshold on $D$ with an optimum diffusion ra…
We study a simple case of the susceptible-weakened-infected-removed model in regular random graphs in a situation where an epidemic starts from a finite fraction of initially infected nodes (seeds). Previous studies have shown that, assuming a single seed, this model exhibits a kind of discontinuous transition at a certain value of infection rate. Performing Monte Carlo simulations and evaluating approximate master equations, we find that the present model has two critical infection rates for the case with a finite seed fraction. At the first critical rate the system shows a percolation transi…
Identifying super-spreaders in epidemics is important to suppress the spreading of disease especially when the medical resource is limited.In the modern society, the information on epidemics transmits swiftly through various communication channels which contributes much to the suppression of epidemics. Here we study on the identification of super-spreaders in the information-disease coupled spreading dynamics. Firstly, we find that the centralities in physical contact layer are no longer effective to identify super-spreaders in epidemics, which is due to the suppression effects from the inform…
We investigate the standard susceptible-infected-susceptible model on a random network to study the effects of preference and geography on diseases spreading. The network grows by introducing one random node with $m$ links on a Euclidean space at unit time. The probability of a new node $i$ linking to a node $j$ with degree $k_j$ at distance $d_{ij}$ from node $i$ is proportional to $k_{j}^{A}/d_{ij}^{B}$, where $A$ and $B$ are positive constants governing preferential attachment and the cost of the node-node distance. In the case of A=0, we recover the usual epidemic behavior with a critical …
Recently, many empirical studies uncovered that animal foraging, migration and human traveling obey Levy flights with an exponent around -2. Inspired by the deluge of H1N1 this year, in this paper, the effects of Levy flights' mobility pattern on epidemic spreading is studied from a network perspective. We construct a spatial weighted network which possesses Levy flight spatial property under a restriction of total energy. The energy restriction is represented by the limitation of total travel distance within a certain time period of an individual. We find that the exponent -2 is the epidemic …
In spite of the extensive previous efforts on traffic dynamics and epidemic spreading in complex networks, the problem of traffic-driven epidemic spreading on {\em correlated} networks has not been addressed. Interestingly, we find that the epidemic threshold, a fundamental quantity underlying the spreading dynamics, exhibits a non-monotonic behavior in that it can be minimized for some critical value of the assortativity coefficient, a parameter characterizing the network correlation. To understand this phenomenon, we use the degree-based mean-field theory to calculate the traffic-driven epid…
Many progresses in the understanding of epidemic spreading models have been obtained thanks to numerous modeling efforts and analytical and numerical studies, considering host populations with very different structures and properties, including complex and temporal interaction networks. Moreover, a number of recent studies have started to go beyond the assumption of an absence of coupling between the spread of a disease and the structure of the contacts on which it unfolds. Models including awareness of the spread have been proposed, to mimic possible precautionary measures taken by individual…
Most studies of disease spreading consider the underlying social network as obtained without the contagion, though epidemic influences people's willingness to contact others: A "friendly" contact may be turned to "unfriendly" to avoid infection. We study the susceptible-infected disease-spreading model on signed networks, in which each edge is associated with a positive or negative sign representing the friendly or unfriendly relation between its end nodes. In a signed network, according to Heider's theory, edge signs evolve such that finally a state of structural balance is achieved, correspo…
Nowadays, the emergence of online services provides various multi-relation information to support the comprehensive understanding of the epidemic spreading process. In this Letter, we consider the edge weights to represent such multi-role relations. In addition, we perform detailed analysis of two representative metrics, outbreak threshold and epidemic prevalence, on SIS and SIR models. Both theoretical and simulation results find good agreements with each other. Furthermore, experiments show that, on fully mixed networks, the weight distribution on edges would not affect the epidemic results …
Wireless networks are commonly used in public spaces, universities and public institutions and provide accurate and easily accessible information to monitor the mobility and behavior of users. Following the application of containment measures during the recent pandemic, we analyse extensive data from the WiFi network in a University Campus in Italy during three periods, corresponding to partial lockdown, partial opening, and almost complete opening. We measure the probability distributions of groups and link activation at Wi-Fi Access Points, investigating how different areas are used in the p…
The impact that information diffusion has on epidemic spreading has recently attracted much attention. As a disease begins to spread in the population, information about the disease is transmitted to others, which in turn has an effect on the spread of disease. In this paper, using empirical results of the propagation of H7N9 and information about the disease, we clearly show that the spreading dynamics of the two-types of processes influence each other. We build a mathematical model in which both types of spreading dynamics are described using the SIS process in order to illustrate the influe…
This study is concerned with the dynamical behaviors of epidemic spreading over a two-layered interconnected network. Three models in different levels are proposed to describe cooperative spreading processes over the interconnected network, wherein the disease in one network can spread to the other. Theoretical analysis is provided for each model to reveal that the global epidemic threshold in the interconnected network is not larger than the epidemic thresholds for the two isolated layered networks. In particular, in an interconnected homogenous network, detailed theoretical analysis is prese…
In this paper, we investigate the epidemic spreading for SIR model in weighted scale-free networks with nonlinear infectivity, where the transmission rate in our analytical model is weighted. Concretely, we introduce the infectivity exponent $α$ and the weight exponent $β$ into the analytical SIR model, then examine the combination effects of $α$ and $β$ on the epidemic threshold and phase transition. We show that one can adjust the values of $α$ and $β$ to rebuild the epidemic threshold to a finite value, and it is observed that the steady epidemic prevalence $R$ grows in an exponential form …
Although suppressing the spread of a disease is usually achieved by investing in public resources, in the real world only a small percentage of the population have access to government assistance when there is an outbreak, and most must rely on resources from family or friends. We study the dynamics of disease spreading in social-contact multiplex networks when the recovery of infected nodes depends on resources from healthy neighbors in the social layer. We investigate how degree heterogeneity affects the spreading dynamics. Using theoretical analysis and simulations we find that degree heter…
We investigate the spread of diseases, computer viruses or information on complex networks and also immunization strategies to prevent or control the spread. When an entire population cannot be immunized and the effect of immunization is not perfect, we need the targeted immunization with immunization rate. Under such a circumstance we calculate epidemic thresholds for the SIR and SIS epidemic models. It is shown that, in scale-free networks, the targeted immunization is effective only if the immunization rate is equal to one. We analyze here epidemic spreading on directed complex networks, bu…
The interplay between traffic dynamics and epidemic spreading on complex networks has received increasing attention in recent years. However, the control of traffic-driven epidemic spreading remains to be a challenging problem. In this Brief Report, we propose a method to suppress traffic-driven epidemic outbreak by properly removing some edges in a network. We find that the epidemic threshold can be enhanced by the targeted cutting of links among large-degree nodes or edges with the largest algorithmic betweeness. In contrast, the epidemic threshold will be reduced by the random edge removal.…
A configuration model for epidemic spread, based on a scale-free network, is introduced. We obtain the analytical solutions describing both unstable and stable dynamics of the epidemic spreading, and demonstrate how these regimes can interchange during the epidemic. We apply the model to the COVID-19 case and demonstrate the predictive features of our model.
We propose a targeted intervention protocol where recovery is restricted to individuals that have the least number of infected neighbours. Our recovery strategy is highly efficient on any kind of network, since epidemic outbreaks are minimal when compared to the baseline scenario of spontaneous recovery. In the case of spatially embedded networks, we find that an epidemic stays strongly spatially confined with a characteristic length scale undergoing a random walk. We demonstrate numerically and analytically that this dynamics leads to an epidemic spot with a flat surface structure and a radiu…
We study Susceptible-Exposed-Asymptomatic-Infectious-Recovered (SEAIR) epidemic spreading model of COVID-19. It captures two important characteristics of the infectiousness of COVID-19: delayed start and its appearance before onset of symptoms, or even with total absence of them. The model is theoretically analyzed in continuous-time compartmental version and discrete-time version on random regular graphs and complex networks. We show analytically that there are relationships between the epidemic thresholds and the equations for the susceptible populations at the endemic equilibrium in all thr…
This article reviews and presents various solved and open problems in the development, analysis, and control of epidemic models. We are interested in presenting a relatively concise report for new engineers looking to enter the field of spreading processes on complex networks.
Epidemic spreading on physical contact network will naturally introduce the human awareness information diffusion on virtual contact network, and the awareness diffusion will in turn depress the epidemic spreading, thus forming the competing spreading processes of epidemic and awareness in a multiplex networks. In this paper, we study the competing dynamics of epidemic and awareness, both of which follow the SIR process, in a two-layer networks based on microscopic Markov chain approach and numerical simulations. We find that strong capacities of awareness diffusion and self-protection of indi…
Understanding and predicting outbreaks of contagious diseases are crucial to the development of society and public health, especially for underdeveloped countries. However, challenging problems are encountered because of complex epidemic spreading dynamics influenced by spatial structure and human dynamics (including both human mobility and human interaction intensity). We propose a systematical model to depict nationwide epidemic spreading in Côte d'Ivoire, which integrates multiple factors, such as human mobility, human interaction intensity, and demographic features. We provide insights to …
In this paper, we study the interplay between individual behaviors and epidemic spreading in a dynamical network. We distribute agents on a square-shaped region with periodic boundary conditions. Every agent is regarded as a node of the network and a wireless link is established between two agents if their geographical distance is less than a certain radius. At each time, every agent assesses the epidemic situation and make decisions on whether it should stay in or leave its current place. An agent will leave its current place with a speed if the number of infected neighbors reaches or exceeds…
Just like the degrees of human and animal interaction networks, the distribution of the times between interactions is known to often be right-skewed and fat-tailed. Both these distributions affect epidemic dynamics strongly, but, as we show in this Letter, for very different reasons. Whereas the high degrees of the tail are critical for facilitating epidemics, it is the small interevent times that control the dynamics of epidemics. We investigate this effect both analytically and numerically for different versions of the Susceptible-Infected-Recovered model on different types of networks.
Random walk is one of the basic mechanisms found in many network applications. We study the epidemic spreading dynamics driven by biased random walks on complex networks. In our epidemic model, each time infected nodes constantly spread some infected packets by biased random walks to their neighbor nodes causing the infection of the susceptible nodes that receive the packets. An infected node get recovered from infection with a fixed probability. Simulation and analytical results on model and real-world networks show that the epidemic spreading becomes intense and wide with the increase of del…
In this paper, we study the interplay between the epidemic spreading and the diffusion of awareness in multiplex networks. In the model, an infectious disease can spread in one network representing the paths of epidemic spreading (contact network), leading to the diffusion of awareness in the other network (information network), and then the diffusion of awareness will cause individuals to take social distances, which in turn affects the epidemic spreading. As for the diffusion of awareness, we assume that, on the one hand, individuals can be informed by other aware neighbors in information ne…
This paper presents a discrete time probabilistic dynamic for simulating a contact-based epidemic spreading based on discrete time Markov chain process, in particular the attention is addressed to the susceptible-infectious-removed (SIR) model and the phase diagram of such model will be presented. Then, this report presents the set of equations that represent the optimal control strategies, by the means of Pontryagin's maximum principle, in two different cases a vaccination policy and a combined vaccination-hospitalization policy and show a numerical simulation, with the standard forward-backw…
The spread of disease through a physical-contact network and the spread of information about the disease on a communication network are two intimately related dynamical processes. We investigate the asymmetrical interplay between the two types of spreading dynamics, each occurring on its own layer, by focusing on the two fundamental quantities underlying any spreading process: epidemic threshold and the final infection ratio. We find that an epidemic outbreak on the contact layer can induce an outbreak on the communication layer, and information spreading can effectively raise the epidemic thr…
A model for epidemic spreading on rewiring networks is introduced and analyzed for the case of scale free steady state networks. It is found that contrary to what one would have naively expected, the rewiring process typically tends to suppress epidemic spreading. In particular it is found that as in static networks, rewiring networks with degree distribution exponent $γ>3$ exhibit a threshold in the infection rate below which epidemics die out in the steady state. However the threshold is higher in the rewiring case. For $2<γ\leq 3$ no such threshold exists, but for small infection rate the s…
In this work, we aim to understand the influence of the heterogeneity of infection rates on the Susceptible-Infected-Susceptible (SIS) epidemic spreading. Employing the classic SIS model as the benchmark, we study the influence of the independently identically distributed infection rates on the average fraction of infected nodes in the metastable state. The log-normal, gamma and a newly designed distributions are considered for infection rates. We find that, when the recovery rate is small, i.e.\ the epidemic spreads out in both homogeneous and heterogeneous cases: 1) the heterogeneity of infe…
We review and introduce a generalized reaction-diffusion approach to epidemic spreading in a metapopulation modeled as a complex network. The metapopulation consists of susceptible and infected individuals that are grouped in subpopulations symbolising cities and villages that are coupled by human travel in a transportation network. By analytic methods and numerical simulations we calculate the fraction of infected people in the metaopoluation in the long time limit, as well as the relevant parameters characterising the epidemic threshold that separates an epidemic from a non-epidemic phase. W…
We propose a novel SIR epidemic model which is driven by the transmission of infection packets in networks. Specifically, infected nodes generate and deliver infection packets causing the spread of the epidemic, while recovered nodes block the delivery of infection packets, and this inhibits the epidemic spreading. The efficient routing protocol governed by a control parameter $α$ is used in the packet transmission. We obtain the maximum instantaneous population of infected nodes, the maximum population of ever infected nodes, as well as the corresponding optimal $α$ through simulation. We fin…
