arXivDaily arXiv每日学术速递 周一至周五更新
arXiv 1105.5545physics.soc-phcond-mat.stat-mechcs.SI
收藏

网络上流行病的竞争激活机制

Competing activation mechanisms in epidemics on networks

Claudio Castellano, Romualdo Pastor-Satorras

摘要:

以前的普遍观点认为,异质网络中的流行病活动是由连接数量最多的枢纽控制的,而最近的研究指出,网络最里面的密集核心在维持流行病过程中发挥着作用。在这里,我们表明,负责传播的机制取决于该过程的性质。具有暂时状态的流行病是由最内层的核心推动的。首先,允许稳定状态的流行病呈现出双重场景,其中要么枢纽独立地维持活动并将其传播到系统的其余部分,要么最内部的网络核心集体进入活动状态,在全球范围内维持它。在不相关的网络中,如果度分布以大于5/2的指数衰减,则前者机制占主导地位,否则后者占主导地位。在真实网络中普遍存在的拓扑相关性可能会扰乱这一图景,混淆这两种机制的作用。

英文摘要:

In contrast to previous common wisdom that epidemic activity in heterogeneous networks is dominated by the hubs with the largest number of connections, recent research has pointed out the role that the innermost, dense core of the network plays in sustaining epidemic processes. Here we show that the mechanism responsible of spreading depends on the nature of the process. Epidemics with a transient state are boosted by the innermost core. Contrarily, epidemics allowing a steady state present a dual scenario, where either the hub independently sustains activity and propagates it to the rest of the system, or, alternatively, the innermost network core collectively turns into the active state, maintaining it globally. In uncorrelated networks the former mechanism dominates if the degree distribution decays with an exponent larger than 5/2, and the latter otherwise. Topological correlations, rife in real networks, may perturb this picture, mixing the role of both mechanisms.

机构、总结、中文摘要均由AI分析而来,可能存在错误,请以论文正文为准。

↑