β-neurexin/neuroligin-1神经元间突触内粘附对量子脑动力学至关重要
The beta-neurexin/neuroligin-1 interneuronal intrasynaptic adhesion is essential for quantum brain dynamics
摘要：
在理解大脑动力学，特别是它如何产生意识方面，有许多空白区域。量子力学被认为能够解释意识经验之谜，但迄今为止还没有一个既能考虑临床神经学数据又具有一定解释力的模型。本文提出了一种新的模型，在防御的宏观量子事件内和之间的神经细胞。突触β-neurexin/neuroligin-1粘附蛋白复合物被认为不仅是兴奋性神经元能CNS突触的核心，相反，它是一种介导皮质神经元细胞骨架之间缠结的装置。因此，宏观相干量子态可以扩展到整个大脑皮层区域，随后的波函数崩溃可以同时影响数百万神经元中的亚神经元事件。neuroligin-1/β-neurexin/synaptotagmin-1复合物还控制胞吐的过程，并在突触连接的学习依赖性变化期间为逆行信号传递提供了一种有趣而简单的机制。通过胞吐作用驱动神经介质释放的分子机制的简要展望，特别强调振动辅助隧道的可能性。
英文摘要：
There are many blank areas in understanding the brain dynamics and especially how it gives rise to consciousness. Quantum mechanics is believed to be capable of explaining the enigma of conscious experience, however till now there is not good enough model considering both the data from clinical neurology and having some explanatory power. In this paper is presented a novel model in defence of macroscopic quantum events within and between neural cells. The synaptic beta-neurexin/neuroligin-1 adhesive protein complex is claimed to be not just the core of the excitatory glutamatergic CNS synapse, instead it is a device mediating entanglement between the cytoskeletons of the cortical neurons. Thus the macroscopic coherent quantum state can extend throughout large brain cortical areas and the subsequent collapse of the wavefunction could affect simultaneously the subneuronal events in millions of neurons. The neuroligin-1/beta-neurexin/synaptotagmin-1 complex also controls the process of exocytosis and provides an interesting and simple mechanism for retrograde signalling during learning-dependent changes in synaptic connectivity. A brief outlook of the molecular machinery driving neuromediator release through exocytosis is provided with particular emphasis on the possibility for vibrationally-assisted tunneling.
机构、总结、中文摘要均由AI分析而来，可能存在错误，请以论文正文为准。
