Psychological effect can lead to bistability in epidemics

Antivax movement and epidemic spreading in the era of social networks: Nonmonotonic effects, bistability and network segregation

In this work, we address a multicoupled dynamics on complex networks with tunable structural segregation. Specifically, we work on a networked epidemic spreading under a vaccination campaign with agents in favor and against the vaccine. Our results show that such coupled dynamics exhibits a myriad of phenomena such as nonequilibrium transitions accompanied by bistability. Besides we observe the emergence of an intermediate optimal segregation level where the community structure enhances negative opinions over vaccination but counterintuitively hinders - rather than favoring - the global diseas…

具有免疫衰减的简单传染病模型中的真实与虚假双稳态

Genuine and spurious bistability in a simple epidemic model with waning immunity

本研究针对感染年龄结构、免疫衰减的传染病模型，结合ODE近似与数值分岔方法分析伽马分布感染持续时间的影响，首次发现仅免疫衰减下的双稳态，揭示分布结构的关键作用及房室模型的局限性。

传染病动力学中三临界行为

Tricritical behavior in epidemic dynamics with vaccination

该研究揭示了疫苗-传染病模型中三临界行为的相变特性及临界现象，表明其属于平均场三临界定向渗透的普遍类别。

具有两级意识的流行病模型中极限环的鞍结分岔

Saddle-node bifurcation of limit cycles in an epidemic model with two levels of awareness

该研究针对含两级意识的非线性流行病模型，通过Poincaré映射分析极限环的鞍结分岔与稳定性，推导地方病平衡点存在条件，并用低输入病例下的随机模拟验证了双稳态现象。

Memory-induced complex contagion in epidemic spreading

Albeit epidemic models have evolved into powerful predictive tools for the spread of diseases and opinions, most assume memoryless agents and independent transmission channels. We develop an infection mechanism that is endowed with memory of past exposures and simultaneously incorporates the joint effect of multiple infectious sources. Analytic equations and simulations of the susceptible-infected-susceptible model in unstructured substrates reveal the emergence of an additional phase that separates the usual healthy and endemic ones. This intermediate phase shows fundamentally distinct charac…

SIS流行病中由观点驱动的风险感知与反应

Opinion-driven risk perception and reaction in SIS epidemics

本文构建观点驱动接触率的SIS耦合模型，证明高低基线传染性两类参数区间及双稳态，并揭示风险寻求、风险规避和高反应性对感染水平及根除的影响。