~（26）Al衰变的人类学与生物同手性
Anthropics of Aluminum-26 Decay and Biological Homochirality
摘要：
最近的实验结果恢复了生物分子同手性起源于β衰变的假设的可行性。再加上这一过程发生在地外的暗示表明铝-26是最有可能的来源。如果是真的，那么它的适当性在很大程度上取决于这种衰变的半衰期和能量。要求这种机制成立，对多个参数的人类允许范围提出了新的限制，包括电子质量、上下夸克质量之差、精细结构常数和电弱尺度。这些新的粒子质量的约束比以前发现的更严格，然而，允许区域的一个边缘几乎退化与现有的限制，其中，使用这里所谓的“非重合危险的原则”，被认为是一个强有力的指标，精细结构常数必须是一个环境参数在多重宇宙。
英文摘要：
Results of recent experiment reinstate feasibility to the hypothesis that biomolecular homochirality originates from beta decay. Coupled with hints that this process occurred extraterrestrially suggests aluminum-26 as the most likely source. If true, then its appropriateness is highly dependent on the half-life and energy of this decay. Demanding that this mechanism hold places new constraints on the anthropically allowed range for multiple parameters, including the electron mass, difference between up and down quark masses, the fine structure constant, and the electroweak scale. These new constraints on particle masses are tighter than those previously found. However, one edge of the allowed region is nearly degenerate with an existing bound, which, using what is termed here as `the principle of noncoincident peril', is argued to be a strong indicator that the fine structure constant must be an environmental parameter in the multiverse.
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